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Prevacid (Lansoprazole)
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Prevacid

Generic Prevacid helps with different acid-related disorders. It treats heartburn by decreasing the amount of acid produced in the stomach. Generic Prevacid can also help cure damage which acid can make to your esophagus. Moreover, Generic Prevacid helps cure duodenal ulcers (ulcers in the first part of the intestine), gastric ulcers (stomach ulcers) and stomach ulcers as a result of continuous use of pain medications called NSAIDs, nonsteroidal anti-inflammatory drugs (aspirin, ibuprofen and naproxen).

Other names for this medication:
Agopton, Amarin, Anzo, Anzoprol, Aprazol, Aslan, Bal-lanz, Bamalite, Betalans, Biolanz, Bivilans, Bylans, Chexid, Compraz, Dakar, Degastrol, Digest, Epicur, Ermes, Estomil, Eudiges, Frilans, Fudermex, Gastrazol, Gastrex, Gastribien, Gastride, Gastrolan, Gastroliber, Gastropec, Helicol, Ilsatec, Imidex, Inhipraz, Iniprazol, Interlansil, Keval, Lacopen, Lamp, Lan, Lancap, Lancibay, Lancid, Lanciprol, Lancus, Lanfast, Lanobax, Lanodizol, Lanopra, Lanoz, Lanpo, Lanpracid, Lanpro, Lanprol, Lanproton, Lans, Lansacid, Lansazol, Lansec, Lanser, Lansina, Lanso, Lanso-q, Lansobene, Lansodin, Lansofast, Lansogamma, Lansogen, Lansohexal, Lansol, Lansoloc, Lansomid, Lansone, Lansopep, Lansopral, Lansoprazol, Lansoprazola, Lansoprazolum, Lansopril, Lansoprol, Lansoptol, Lansoquilab, Lansor, Lansoral, Lansosiga, Lansotop, Lansotrent, Lansovax, Lansox, Lanspep, Lanspro, Lantera, Lantid, Lanton, Lanximed, Lanz, Lanzap, Lanzedin, Lanzet, Lanziop, Lanzo, Lanzogastro, Lanzohess, Lanzol, Lanzolab, Lanzonium, Lanzopral, Lanzoprazol, Lanzor, Lanzostad, Lanzul, Lapol, Lapraz, Laprazol, Laproton, Laprotone, Larona, Lasgan, Lasobix, Lasopran, Lasoprol, Lasovac, Laz, Lazol, Leedom, Levant, Lexid, Lezo cap, Limpidex, Linibyn, Liza, Liza-d, Loprezol, Lupizole, Medamarin, Mesactol, Monolitum, Nufaprazol, Ogast, Ogasto, Ogastoro, Ogastro, Opagis, Opelansol, Opiren, Palatrin, Peptazole, Prazex, Prazotec, Prezal, Prilosan, Pro ulco, Propilan, Propump, Prosogan, Protica, Protogut, Protolan, Protoner, Protonexa, Rapilazole, Rarpezol, Razolager, Reflan, Refluxon, Refluyet, Renazol, Safemar, Selanz, Solans, Solox, Sopralan, Splanz, Stanzome, Taiproton, Takepron, Tapizol, Taquidine, Tersen, Trogas, Ulceran, Uldapril, Ulpax, Ultrazole, Vogast, Zalanzo, Zapacid, Zolt, Zomel, Zoprol, Zoton, Zotrole

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Also known as:  Lansoprazole.

Description

Generic Prevacid helps with different acid-related disorders.

Generic Prevacid helps with different acid-related disorders. It treats heartburn by decreasing the amount of acid produced in the stomach. Generic Prevacid can also help cure damage which acid can make to your esophagus. Moreover, Generic Prevacid helps cure duodenal ulcers (ulcers in the first part of the intestine), gastric ulcers (stomach ulcers) and stomach ulcers as a result of continuous use of pain medications called NSAIDs, nonsteroidal anti-inflammatory drugs (aspirin, ibuprofen and naproxen).

Prevacid is also known as Lansoprazole, Fenzer, Lan, Helicid, Zoton, Inhibitol, Takepron.

Generic Prevacid helps with different acid-related disorders.

Brand name of Generic Prevacid is Prevacid.

Dosage

Generic Prevacid is available in 3 forms such as Generic Prevacid Capsules (15 mg, 30 mg), Generic Prevacid SoluTab Orally Disintegrating Tablets and Generic Prevacid for Oral Suspension.

Generic Prevacid Capsules

Generic Prevacid Capsules should be taken before eating. If you can't swallow you can open a capsule and sprinkl into 1/4 cup of tomato, apple or orange juice, or on a tablespoon of applesauce, Ensure pudding, cottage cheese, yogurt or strained pears. Make sure to drink and eat food sprinkled with Generic Prevacid right away and be careful not to crush or chew the capsules or the granules. If you want to sprinkle Generic Prevacid granules, you should only do this in the juices and foods mantioned above.

Generic Prevacid SoluTab Orally Disintegrating Tablets

It is not necessary to take Generic Prevacid SoluTab with water. It is easy to swallow it without any water. Generic Prevacid SoluTab has a strawberry flavor.

Generic Prevacid for Oral Suspension

The contents of a packet should be mixed with two tablespoons of water, stirred and drunk immediately. Generic Prevacid for Oral Suspension should not be given through a feeding tube and should not be mix with other liquids or foods. Generic Prevacid for Oral Suspension has a strawberry flavor. 24 hours can be enough for Generic Prevacid to relieve heartburn and many other symptoms of acid reflux disease.

If you want to achieve most effective results do not stop taking Generic Prevacid suddenly.

Overdose

If you overdose Generic Prevacid and you don't feel good you should visit your doctor or health care provider immediately.

Storage

Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Prevacid are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Do not take Generic Prevacid if you are allergic to Generic Prevacid components.

Be careful with Generic Prevacid if you're pregnant or you plan to have a baby.

Generic Prevacid can be taken by children aged 1-17 for GERD.

Do not take Generic Prevacid if you have creatinine clearance less than 30 mL/min.

Do not use Generic Prevacid if you take cisapride, pimozide, astemizole, terfenadine, ergotamine or dihydroergotamine.

Do not use potassium supplements or salt substitutes.

If you want to achieve most effective results without any side effects it is better to avoid alcohol.

Be very careful when you are driving machine.

Do not stop taking Generic Prevacid suddenly.

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The quality attributes of extemporaneous delayed-release liquid formulations of lansoprazole for oral administration were evaluated.

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Lansoprazole safely and effectively reduces duodenal ulcer recurrence and ulcer-related symptoms.

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Chronic gastritis induced by Helicobacter pylori is the strongest known risk factor for gastric adenocarcinoma, yet the effects of bacterial eradication on carcinogenesis remain unclear. Animal models provide important insights into factors that are involved in gastric carcinogenesis, and we previously utilized such a model to demonstrate that an in vivo-adapted H. pylori strain, 7.13, rapidly and reproducibly induces inflammation-mediated gastric carcinoma. In the current study, we used this bacterial strain as a prototype to define the role of targeted antimicrobial therapy in gastric carcinogenesis. Mongolian gerbils were infected with H. pylori for 4 or 8 weeks, treated with antimicrobial agents or vehicle, and then euthanized at 8 weeks after the completion of therapy. All infected gerbils developed gastritis; however, inflammation was significantly attenuated in animals receiving antimicrobial therapy. Gastric dysplasia or cancer developed in >60% of the gerbils that remained persistently colonized with H. pylori, but in none of the animals treated with antibiotics following 4 weeks of infection. Infection with H. pylori for 8 weeks prior to therapy resulted in an attenuation, but not complete prevention, of pre-malignant and malignant lesions. Similarly, antibiotic therapy initiated at 4, but not 8, weeks after H. pylori challenge significantly reduced expression of the Th1 pro-inflammatory cytokine interferon-gamma within colonized gastric mucosa. These results indicate that treatment of H. pylori in this model decreases the incidence and severity of lesions with carcinogenic potential. The effectiveness of eradication is dependent upon the timing of intervention, providing insights into mechanisms that may regulate the development of malignancies arising within the context of inflammatory states.

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Prospective study to assess the accuracy of the urea breath test during the first days of therapy with proton pump inhibitors.

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Gastroesophageal reflux is common among patients with postnasal drainage. We investigated whether proton pump inhibitor therapy improved symptoms in patients with postnasal drainage without sinusitis or allergies.

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Background Tranexamic acid reduces haemorrhage through its antifibrinolytic effects. In a previous version of the present review, we found that tranexamic acid may reduce mortality. This review includes updated searches and new trials.Objectives To assess the effects of tranexamic acid versus no intervention, placebo or other antiulcer drugs for upper gastrointestinal bleeding.Search methods We updated the review by performing electronic database searches (Cochrane Central Register of Controlled Trials (CENTRAL),MEDLINE, EMBASE, Science Citation Index) and manual searches in July 2014.Selection criteriaRandomised controlled trials, irrespective of language or publication status.Data collection and analysis We used the standard methodological procedures of the The Cochrane Collaboration. All-cause mortality, bleeding and adverse events were the primary outcome measures. We performed fixed-effect and random-effects model meta-analyses and presented results as risk ratios (RRs) with 95% confidence intervals (CIs) and used I² as a measure of between-trial heterogeneity. We analysed tranexamic acid versus placebo or no intervention and tranexamic acid versus antiulcer drugs separately. To analyse sources of heterogeneity and robustness of the overall results, we performed subgroup, sensitivity and sequential analyses.Main results We included eight randomised controlled trials on tranexamic acid for upper gastrointestinal bleeding. Additionally, we identified one large ongoing pragmatic randomised controlled trial from which data are not yet available. Control groups were randomly assigned to placebo (seven trials) or no intervention (one trial). Two trials also included a control group randomly assigned to antiulcer drugs(lansoprazole or cimetidine). The included studies were published from 1973 to 2011. The number of participants randomly assigned ranged from 47 to 216 (median 204). All trials reported mortality. In total, 42 of 851 participants randomly assigned to tranexamic acid and 71 of 850 in the control group died (RR 0.60, 95% CI 0.42 to 0.87; P value 0.007; I² = 0%). The analysis was not confirmed when all participants in the intervention group with missing outcome data were included as treatment failures, or when the analysis was limited to trials with low risk of attrition bias. Rebleeding was diagnosed for 117 of 826 participants in the tranexamic acid group and for 146 of 825 participants in the control group (RR 0.80, 95% CI 0.64 to 1.00; P value 0.07; I² = 49%).We were able to evaluate the risk of serious adverse events on the basis of only four trials. Our analyses showed 'no evidence of a difference between tranexamic acid and control interventions regarding the risk of thromboembolic events.’ Tranexamic acid appeared to reduce the risk of surgery ina fixed-effect meta-analysis (RR 0.73, 95% CI 0.56 to 0.95), but this result was no longer statistically significant in a random-effects meta-analysis (RR 0.61, 95% CI 0.35 to 1.04; P value 0.07). No difference was apparent between tranexamic acid and placebo in the assessment of transfusion (RR 1.02, 95% CI 0.94 to 1.11; I² = 0%), and meta-analyses that compared tranexamic acid versus antiulcer drugs did not identify beneficial or detrimental effects of tranexamic acid for any of the outcomes assessed.Authors' conclusions This review found that tranexamic acid appears to have a beneficial effect on mortality, but a high dropout rate in some trials means that we cannot be sure of this until the findings of additional research are published. At the time of this update in 2014, one large study(8000 participants) is in progress, so this review will be much more informative in a few years. Further examination of tranexamic acid would require inclusion of high-quality randomised controlled trials. Timing of randomisation is essential to avoid attrition bias and to limit the number of withdrawals. Future trials may use a pragmatic design and should include all participants with suspected bleeding or with endoscopically verified bleeding, as well as a tranexamic placebo arm and co-administration of pump inhibitors and endoscopic therapy. Assessment of outcome measures in such studies should be clearly defined. Endoscopic examination with appropriate control of severe bleeding should be performed, as should endoscopic verification of clinically significant rebleeding. In addition, clinical measures of rebleeding should be included. Other important outcome measures include mortality (30-day or in-hospital), need for emergency surgery or blood transfusion and adverse events (major or minor).

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Pretreatment with H2RA had no significant influence on the efficacy of H. pylori eradication therapy.

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305 patients were treated with a regimen containing 400 mg of clarithromycin (C400 group), and 296 patients with a regimen containing 800 mg (C800 group). Overall cure rates between the two groups were not significantly different, but the cure rate in the C800 group was significantly better than that in the C400 group among patients infected with clarithromycin-sensitive strains (p = 0.03). This difference could be attributed to differences among smokers versus non-smokers: the cure rate among smokers in the C800 group (91.0%) was better than that in the C400 group (80.0%, p = 0.003).

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H pylori eradication therapy for peptic ulcer patients with a mean age of 52.9 years at registration did not significantly decrease the incidence of gastric cancer.

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Due to the prevalence of both gastrointestinal and cardiovascular diseases, it is likely that patients may be coprescribed gastric parietal cell proton pump inhibitors and beta-adrenergic antagonists. Therefore, the objectives of this phase I study were to assess the potential effects of the coadministration of lansoprazole on the pharmacokinetics of propranolol and to evaluate the safety of propranolol with concomitant lansoprazole dosing. In a double-blind fashion, 18 healthy male nonsmokers were initially randomized to receive either 60 mg oral lansoprazole, each morning for 7 days, or an identical placebo (period 1). On day 7, all subjects were concomitantly administered oral propranolol, 80 mg. After a minimum of 1 week following the last dose of either lansoprazole or placebo, subjects were crossed over to the opposite treatment for another 7 days (period 2). Subjects were again administered oral propranolol on day 7. During both treatment periods, blood samples for the determination of plasma propranolol and 4-hydroxy-propranolol were obtained just before the dose and at 0.5, 1, 2, 3, 4, 6, 8 12, 16, 20, and 24 hours postdose. Plasma propranolol and 4-hydroxy-propranolol concentrations were determined by using HPLC with fluorescence detection. The Cmax, tmax, AUC0-infinity, and t1/2 values for propranolol, as well as the AUC0-infinity for 4-hydroxy-propranolol, were calculated and compared between the lansoprazole and placebo regimens. The mean age of the 15 subjects who successfully completed the study was 31 years (range: 24-38 years), and their average weight was 174.8 pounds (range: 145-203 pounds). There were no statistically significant differences between the lansoprazole and placebo regimens for the propranolol Cmax, tmax, AUC0-infinity, and t1/2 values. Also, there were no statistically significant differences between regimens for the 4-OH-propranolol AUC0-infinity. Safety evaluations, which included adverse events, vital signs, clinical laboratory determinations, ECG, and physical examinations, revealed no unexpected clinically significant findings and did not suggest a drug-drug interaction. In conclusion, lansoprazole does not significantly alter the pharmacokinetics of propranolol, suggesting that it does not interact with the CYP2D6- or CYP2C19-mediated metabolism of propranolol. Modification of a propranolol dosage regimen in the presence of lansoprazole is not indicated, based on the pharmacokinetic analysis and the lack of a clinically significant alteration in the pharmacodynamic response.

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prevacid 80 mg 2016-08-13

Proton pump inhibitors including omeprazole and lansoprazole inhibit gastric acid secretion by selectively and non-competitively inactivating the H+, K+ ATPase molecules of the parietal cell, but possibly only those that are actively secreting acid. This might imply that Bactroban Pill stimulation of acid secretion by a meal is necessary for optimal inhibition of gastric secretion.

prevacid go generic 2015-12-10

During a median follow-up of 24 weeks, 4 (3.7%, 95% confidence interval [CI] 0.0%-7.3%) patients in the celecoxib group, compared with 7 patients (6.3%, 95% CI 1.6%-11.1%) in the lansoprazole group, developed recurrent ulcer complications (absolute difference -2.6%; 95% CI for the difference -9.1%-3.7%). Celecoxib was statistically non-inferior to lansoprazole co-therapy in the prevention of recurrent ulcer complications. Concomitant illness (hazard ratio 4.72, 95% CI 1.24-18.18) and age 65 Crestor Tablet Image years or more (hazard ratio 18.52, 95% CI 2.26-142.86) were independent risk factors for ulcer recurrences. Significantly more patients receiving celecoxib (15.0%, 95% CI 9.7-22.5) developed dyspepsia than patients receiving lansoprazole (5.7%, 95% CI 2.8-11.4. P = .02).

prevacid generic otc 2015-06-04

Meta-analysis is a useful tool Terramycin Dosage in obtaining sound data in evidence-based medicine. The guidelines of eradication have been proposed in the I. Maastricht consensus (1996), which was modified in 2000.

prevacid dosage forms 2015-12-02

Expression of IL-8 mRNA was correlated with the endoscopic grade of esophagitis or with inflammatory cell infiltration, but not with the symptoms of the patients. Expression of IL-8 mRNA was also detected in all patients with non-erosive GERD. The level of IL-8 expression in non-erosive GERD was low compared with that in erosive GERD, but was higher than that Lexapro Regular Dosage in normal controls. IL-8 immunostaining was found in the basal layers of the esophageal mucosa. Administration of lansoprazole, a proton-pump inhibitor, decreased both IL-8 mRNA and protein levels in the esophageal mucosa.

prevacid user reviews 2016-12-16

Dexlansoprazole modified release (dexlansoprazole MR) is an orally administered delayed-release formulation of the R-enantiomer of the proton pump inhibitor lansoprazole that is effective in improving the healing of all grades of erosive oesophagitis, maintaining the healing of erosive oesophagitis and in the treatment of symptomatic non-erosive reflux disease (NERD). In two large, identical, 8-week, randomized, double-blind, multicentre phase III trials, dexlansoprazole MR 60 mg once daily achieved complete healing in >or=92% of patients with all grades of erosive oesophagitis (primary endpoint) and was noninferior to lansoprazole 30 mg once daily using life-table analysis. Moreover, in a randomized, double-blind, multicentre phase III trial in patients with healed erosive oesophagitis, dexlansoprazole MR 30 mg once daily was significantly more effective than placebo in maintaining healing following 6 months' treatment (primary endpoint). In addition, the proportion of 24-hour heartburn-free days (primary endpoint) was significantly greater in recipients of dexlansoprazole MR 30 mg once daily than in recipients of placebo following 4 weeks' treatment in a large, randomized, double-blind Voltaren Brand Name , multicentre phase III trial in patients with NERD. Dexlansoprazole MR 30 or 60 mg once daily was generally well tolerated in a pooled analysis of clinical trials of up to 12 months' duration.

prevacid heartburn medicine 2017-12-27

Adolescents between 12 and 17 years of age with esophagitis were enrolled in this open-label trial and treated with lansoprazole 15 mg (non-erosive) or 30 mg (erosive) once daily for 8 weeks. If unhealed at week 8, those with erosive esophagitis were treated Asacol Generic Name with an additional 4 weeks of lansoprazole 30 mg once daily.

prevacid tabs 2015-07-17

We conducted a double-blind, randomized, placebo-controlled, multicenter trial comparing the efficacy of 7 days of eradication treatment (lansoprazole 15 mg b.i.d., amoxicillin I g b.i.d., and clarithromycin 500 mg b.i.d.) with a control treatment (lansoprazole 15 mg b.i.d. and placebo) in H. pylori-infected patients with nonulcer dyspepsia. 13C breath tests were performed at baseline and during follow-up. We assessed patient symptoms, health status (based on the SF-12 questionnaire), patient satisfaction, drug consumption, health care Celexa 5 Mg consultation behavior, and absenteeism related to dyspepsia over a 1-yr period.

prevacid 40 mg 2017-07-24

After each tasting, children rated their degree of liking on a five-point facial hedonic scale (5 = like very much, 1 = dislike very much). Product Vasotec Generic Cost preference was recorded after the tasting of both samples.

prevacid dosage infants 2017-09-11

The isolated compounds were identified by spectroscopic means. The gastroprotective effect of the compounds was studied on the HCl/EtOH-induced gastric lesions model in mice. The cytotoxicity of the compounds was assessed on human normal lung fibroblasts (MRC-5) and gastric adenocarcinoma cells (AGS).

prevacid cost 2016-08-08

Thirty-five consecutive adult patients with paroxysmal laryngospasm (LS) and with unimpaired vocal fold mobility were prospectively studied for coexisting gastroesophageal reflux disease (GERD). Nineteen patients reported frequent (>3 episodes a week) LS episodes (FLS patients) and 16 patients reported occasional LS episodes (OLS patients). All patients underwent an extensive otorhinolaryngological (ORL) examination, upper gastrointestinal (GI) endoscopy, ambulatory 24-hr dual-channel esophageal pH monitoring, and esophageal manometry. In addition, a subset of LS patients also underwent ambulatory duodenogastroesophageal reflux (DGER) monitoring. Patients with daily LS used the symptom marker during pH monitoring indicating separate LS episodes. All FLS patients and 14 OLS patients (87%) had a diagnosis of GERD. Only 10 patients (29%) experienced heartburn and/or regurgitation. Compared to OLS patients, FLS patients generally had more severe GERD as indicated by a higher prevalence of a hiatus hernia, higher distal and proximal esophageal acid exposure times, and higher values of DGER. In six FLS patients, 21 LS episodes (91%) occurred simultaneously with acid reflux, indicating a causal association between LS and GER. On antireflux therapy consisting of omeprazole, 20 mg bid, or lansoprazole, 30 mg oid, and lifestyle measures, LS ceased completely in all patients within 6 weeks. The present study not only demonstrates the role of GER in the pathogenesis of LS and the effectiveness of antireflux therapy, but also suggests that LS in adult patients with unimpaired vocal fold mobility might be considered a typical, although most frequently unrecognized, supraesophageal manifestation of GER.

prevacid solutab generic 2015-07-30

Lansoprazole 30 mg, amoxicillin 1000 mg, and tinidazole 500 mg were given twice daily to 39 peptic ulcer patients (26 duodenal and 13 gastric ulcer, mean age 52.4 +/- 15.01) who had H. pylori infection for two weeks. Additional lansoprazole 30 mg daily was given to duodenal and gastric ulcer patients for another two and six weeks respectively. Follow-up gastroduodenoscope was performed at fourth and eighth week and eighth and twelfth week for all duodenal and gastric ulcer patients, respectively. H. pylori status was evaluated by rapid urease test (CLO test) and histology at first and last endoscope. The ulcers were healed at the last endoscopy in 11 (85%) gastric ulcer patients and 24 (92%) duodenal ulcers patients. H. pylori infection was eradicated in 31 patients (79%). Mild side effects were observed in 15 per cent. In conclusion, 2 week regimen of lansoprazole, amoxicillin, and tinidazole triple therapy resulted in a relatively high healing rate of peptic ulcer (90%) and an acceptable eradication rate of H. pylori infection (79%).

prevacid 5 mg 2016-09-23

Patients with a high intragastric bacterial load, as detected by histology (grade 3) or 13C-UBT (delta > 35) may benefit from an extension to 2 wk of triple therapy with lansoprazole, amoxycillin, and clarithromycin.

prevacid infant dosage 2017-05-26

There was no significant difference in the eradication rates between the two groups. In comparison of eradication rates according to PPI, omeprazole- based triple therapy group showed higher eradication rate than other groups in patients with NUD, but not in patients with PUD.

prevacid otc mg 2015-04-13

A total of 92 patients presenting with symptoms of dyspepsia were enrolled in the study. Upper endoscopy was performed on all patients and used to diagnose H. pylori infection according to the presence of characteristic histopathological findings; seventy patients were diagnosed with H. pylori infection and the remaining 22 non-infected patients were classified as healthy controls. H. pylori eradication was accomplished by administering the classical triple therapy drug regimen, consisting of lansoprazole 30 mg bid, amoxicillin 1 g bid, and clarithromycin 500 mg tid for 14 d. The eradication of H. pylori was assessed with C¹⁴-urea breath test, which was performed at eight weeks after treatment. Levels of serum active ghrelin and obestatin were assessed at beginning of the study (prior to treatment) and after eight weeks. The levels were comparatively analyzed between the H. pylori negative control group, the H. pylori eradicated group, and the H. pylori non-eradicated group.

prevacid dose baby 2015-11-28

This Cochrane review aims to provide a robust analysis of currently available pharmacological interventions used to treat children with GOR by assessing all outcomes indicating benefit or harm.