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Ventolin (Albuterol)

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Ventolin is a high-effective medication which is indicated for the relief and prevention of airway obstruction (bronchospasm) in patients with asthma and in patients with exercise-induced asthma. Ventolin can also be used in treating patients with emphysema and chronic bronchitis when their symptoms are related to reversible airway obstruction.

Other names for this medication:
Airet, Airomir, Apo-salvent, Assal, Asthalin, Aurosal, Avedox-fc, Broncovaleas, Ecutamol, Farbutamol, Novo-salmol, Salamol, Salbubronch, Salbutalan, Salbutamol, Salvent, Sultanol, Ventide, Ventodisk, Ventorlin, Volmax

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Also known as:  Albuterol.


Ventolin belongs to a class of drugs known as bronchodilators. Ventolin is indicated for the relief and prevention of airway obstruction (bronchospasm) in patients with asthma and in patients with exercise-induced asthma. Ventolin can also be used in treating patients with emphysema and chronic bronchitis when their symptoms are related to reversible airway obstruction.

Albuterol, the active ingredient in Ventolin is a selective beta-adrenergic bronchodilator used to treat severe acute asthma and chronic bronchospasm caused by other pulmonary obstructive disorders that have not responded to other forms of therapy.

Generic names of Ventolin are Albuterol, Salbutamol.

Ventolin is also known as Albuterol, Salbutamol, Ventorlin, Asthalin, Proventil, ProAir, Salamol, Aerolin.


Follow the directions for using this medicine provided by your doctor. Use Ventolin exactly as directed.

Take this medication by mouth as directed by your doctor.

Do not crush or chew it. Swallow the pill whole. Crushing or chewing Ventolin will negate the delayed release mechanism of the medication.

-The usual effective dose is 4mg, three or four times per day.

-If adequate bronchodilation is not obtained, each single dose may be gradually increased to as much as 8mg.

-Some patients obtain adequate relief with 2 mg three or four times daily.

2 - 6 years: The minimum starting dose is 1mg three times daily. This may be increased to 2mg (1 tablet), three or four times daily.

6 - 12 years: The minimum starting dose is 2mg three times daily. This may be increased to four times daily.

Over 12 years: The minimum starting dose is 2mg three times daily. This may be increased to 4mg (2 tablets), three or four times daily.

In elderly patients or in those known to be usually sensitive to beta-adrenergic stimulant drugs, it is advisable to initiate treatment with 2 mg salbutamol three or four times per day.


If you overdose Ventolin and you don't feel good you should visit your doctor or health care provider immediately.


Store at a room temperature not exceeding 30 degrees C (86 degrees F) away from moisture, light and heat. Throw away the after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Ventolin are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Do not take Ventolin if you are allergic to Ventolin components.

It is not known whether Ventolin will harm an unborn baby. Do not use this medicine without your doctor's advice if you are pregnant or breast-feeding.

Be careful with Ventolin if you have diabetes, heart disease, high blood pressure (hypertension), hyperthyroidism, irregular heart beats (arrhythmias).

Ventolin may make you dizzy. Do not drive, use machinery, or do any activity that requires alertness until you are sure you can perform such activities safely.

Do not stop treatment, even if you are feeling better, unless your doctor tells you to. It may take 2 weeks or longer before you feel the full benefit of the medication.

Avoid alcohol.

Do not stop taking Ventolin suddenly.

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Substituting levalbuterol for racemic albuterol in the ED management of acute asthma significantly reduced the number of hospitalizations.

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Pulmonary function laboratory at university medical center.

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The weight change per actuation and aerosol particle size and number delivered by albuterol metered dose inhalers (MDIs) were measured in a multiplace hyperbaric chamber at pressures ranging from one atmosphere absolute (1 ATA, 0 feet of seawater, fsw, 101 kPa) to three ATA (66 fsw, 304 kPa). Weight change per actuation by CFC (chlorofluorocarbon) and long canister HFA (hydrofluoroalkane) powered MDIs was 13 +/- 1% and 12 +/- 1% less, respectively, at 3 ATA compared to 1 ATA. However, weight change per actuation by short canister HFA MDIs was not significantly changed with pressure. The geometric mean diameters of nano particles from the CFC and short canister HFA MDIs decreased from 50 nm at 0 fsw to 32 nm at 66 fsw whereas the long canister HFA aerosol diameters were not affected. The numbers of nanometer size particles delivered at 66 fsw were only 4-7% of those delivered at 0 fsw for the CFC and long canister HFA MDIs whereas for the short canister MDIs it was 26%. We conclude that the weight change per actuation of albuterol and the sizes and numbers of aerosol particles emitted from albuterol MDIs actuated in a hyperbaric environment vary by canister type.

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In 1997, national guidelines emphasized that inhaled corticosteroids (ICSs) are key therapy for individuals with all classes of persistent asthma, including children.

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Outcome measures included Asthma Quality of Life Questionnaire (AQLQ) global and individual domain scores, FEV1, PEF, nighttime awakenings, asthma symptoms, and supplemental albuterol use. Mean change from baseline for the global and domain AQLQ scores was significantly greater (p < or = 0.005) with salmeterol compared with placebo. At week 12, salmeterol significantly (p < 0.001 compared with placebo) increased mean change from baseline in FEV1, morning and evening PEF, percentage of symptom-free days, percentage of nights with no awakenings due to asthma, and the percentage of days and nights with no supplemental albuterol use. Significant improvements in PEF were observed after treatment with salmeterol regardless of concomitant treatment with theophylline (p < 0.05).

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The effects of frequent high-dose nebulized salbutamol on plasma glucose concentrations were studied in 12 children (mean age 60 months) with acute severe asthma. There was a significant difference between mean initial glucose levels and peak levels. We conclude that hyperglycaemia can be produced in children as a result of nebulized salbutamol.

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Compared to placebo, all active treatments led to significant improvements (p < 0.05) in geometric mean AMP-PC(20): placebo, 42 mg/mL; ML(10), 106 mg/mL; SM(50), 115 mg/mL; ML(10) and SM(50), 183 mg/mL; and ML(10) and SM(100), 247 mg/mL. The effects of montelukast and salmeterol were numerically additive, with ML(10) and SM(100) being significantly different (p < 0.05) from ML(10) alone. For mean FEV(1) and forced expiratory flow rate between 25% and 75% of vital capacity, there were significant differences (p < 0.05) between both combination therapies vs ML(10) alone.

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Twenty Four patients in each group were studied. INSURE failure was seen in16 (66.7 %) and 10 (41.7 %) of normal saline and salbutamol groups respectively (p = 0.082). The duration of NCPAP in control group was 69.5 ± 54.9 h while in Salbutamol group was 51.6 ± 48.7 h (p = 0.316). All of deaths were related to respiratory failure. No differences in mortality or complications of RDS were observed. The duration of hospitalization was longer in control group than interventional group, 28.3 ± 18.1 and 18.6 ± 8.6 days, respectively. (p = 0.047).

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The influence of salbutamol aerosol on heart rate in patients with chronic atrial fibrillation was examined. The study comprised 15 patients (9 women and 6 man) in age 47-79 (mean 60) treated because of chronic obstructive pulmonary disease (COPD) with concomitant atrial fibrillation. The influence of salbutamol on heart rate was investigated during six hours ecg Holter monitoring (3 hours before and 3 hours after using salbutamol). The analysis showed that salbutamol aerosol in two doses increased heart rate calculated as mean heart rate per hour from 3-hours intervals from 4061 congruent to 1022.5 to 4185 congruent to 976 (p greater than 0.05). Mean value of blood pressure in one hour intervals did not change significantly. In patients with concomitant chronic atrial fibrillation and COPD salbutamol increased heart rate but difference between mean values before and after administration was not significant.

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The aim of this study was to investigate whether beta-2-adrenergic stimulation with inhaled salbutamol is therapeutically useful in hyperkalaemia. Ten patients with renal failure and hyperkalaemia (serum potassium concentration greater than 6 mmol l-1) were given 15 mg salbutamol via a nebulizer over a 30-min period. Serum potassium was measured 30, 60, 180 and 360 min thereafter. All patients had end-stage renal failure on chronic hospital haemodialysis. Serum potassium levels decreased significantly from a pretreatment value of 6.5 +/- 0.6 mmol l-1 to 5.6 +/- 0.6 mmol-1 after 30 min, and this level was maintained for 3 h. Six hours after treatment, the serum potassium concentration was 6 +/- 0.7 mmol l-1. There was a modest increase in heart rate and blood glucose level, but otherwise salbutamol was well tolerated and no serious side-effects occurred. It is concluded that the administration of salbutamol by inhalation is a simple, safe and reasonably effective method for treatment of hyperkalaemia in renal failure.

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Within-subject coefficients of variation (WCV) of absolute blood flow responses for each dose of acetylcholine (7.5, 15, 30 micrograms min(-1)) ranged from 24% to 27%, as compared with WCV values of 41% to 62% for the percentage changes in blood flow ratio (infused : noninfused arm). For albuterol (0.3, 1, 3 micrograms min(-1)) the corresponding WCV values were 16% to 19% and 30% to 55% for absolute blood flow and percentage change in blood flow ratio, respectively. WCV for the area under dose-response curve (AUC) for absolute blood flow was 18% and 13% for acetylcholine and albuterol, respectively. Vasodilator responses were similar whether recorded proximal to or at the point of maximal forearm circumference. Distal strain gauge misplacement underestimated responses and the difference was greater for acetylcholine than for albuterol.

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The generic albuterol MDI delivers a quantity of albuterol to the beta(2)-receptor site in the lung that is the bioequivalent to Ventolin. Further, this study reinforces the validity of this statistical methodology for determining in vivo bioequivalence.

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In this study, in children with stable asthma, three consecutive doses of arformoterol (7.5 and 15 µg) and levalbuterol were overall well tolerated. Nonetheless, serum potassium levels demonstrated substantial mean declines after dosing. These findings do not address or support the safety and tolerability of arformoterol use in acute exacerbations of asthma in children.

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Albuterol (2.5 to 10 mg) or placebo was given via nebulizer. Volunteers were studied at two sessions, 1 week apart, using a 6-cm manometry assembly and a low-compliance pneumohydraulic pump. The percentage of lower esophageal sphincter (LES) relaxation, the frequency of transient LES relaxations (TLESRs), and the amplitude, duration, and propagation velocity of esophageal contractions were measured at 5 and 10 cm above the LES. Dependent measures were evaluated using two-way, repeated-measures analysis of variance.

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On four separate occasions the patient was challenged advertently or inadvertently, with sustained-release verapamil. Similar symptoms developed after each challenge and resolved after discontinuing this preparation. Although dyspnea associated with verapamil administration has been reported, this is the first report of an elderly asymptomatic asthmatic patient with hypertension who developed an acute asthma attack following sustained-release verapamil administration.

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ventolin capsule 2016-07-24

Clinicians are faced with an increasingly difficult choice regarding the optimal bronchodilator for patients with chronic obstructive pulmonary disease (COPD) given the number of new treatments. The objective of this study is Augmentin And Alcohol to evaluate the comparative efficacy of indacaterol 75/150/300 μg once daily (OD), glycopyrronium bromide 50 μg OD, tiotropium bromide 18 μg/5 μg OD, salmeterol 50 μg twice daily (BID), formoterol 12 μg BID, and placebo for moderate to severe COPD.

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A total of 3,503 subjects were identified based on their index medication: FSC (996), FP+SAL (259), FP+MON (101), FP (1254) and MON (893). Mean number of prescription refills for FSC (3.98) was significantly higher than FP (2.29) and the FP component of FP+SAL (2.36), and FP+MON (2.15), P<0.05. No significant differences were Buy Vermox Online observed between FSC and MON fill rates (4.33). Mean number of treatment days was greater for FSC compared to FP, FP+SAL, and FP+MON (P<0.0001).

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In this review the authors show that (R Indocin 25 Mg )-salbutamol alone (generically known as levosalbutamol) provides beneficial β(2)-agonist effects at a cellular level and in experimental models of airways disease. In addition the authors demonstrate that (S)-salbutamol opposes the desirable effects of (R)-salbutamol and can actually cause features of asthma and COPD in vitro and in experimental asthma.

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Previous studies have suggested that respiratory infection during childhood is associated with respiratory disease in adulthood, but the link is unclear because of retrospective ascertainment of childhood infection, selection bias, and confounding Imitrex 2nd Dose factors.

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A new method for the determination of salbutamol has been developed using fullerene C60-modified glassy carbon electrode and validated using GC-MS. The presence of graphite and metallic impurities in C60 are found to diminish the peak. The oxidation of salbutamol was observed in a single well-defined, diffusion-controlled process using square wave voltammetry. The peak potential of oxidation peak was dependent on pH and determination was carried out at physiological pH 7.4. The peak current versus concentration plot was linear in the range 100-2000 ng/ml of salbutamol. The detection limit was found to be 40 ng/ml. The determination of salbutamol was carried out in human blood and urine samples and common interferents such as dopamine, ascorbic acid and uric acid do not interfere. The method proved to be specific, rapid, and accurate and can be easily applied for detecting cases of doping. A cross-validation of the observed results with Levitra Generic Online GC-MS indicated a good agreement.

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Because published criteria for bronchodilator responsiveness are based on population variability and compare only the single best measures before and after intervention, we hypothesized that the variability of the FEV1 and FEV3 of each patient would Amaryl Tabs better determine that patient's responsiveness.

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We report a case of complex Zithromax 250mg Capsules and dramatic sleepwalking behavior in a 26-year-old adult male who tied his 4-month-old daughter to the clothesline in the attic of his house.

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Patients were randomized to formoterol (n = 5,367), placebo (n = 2,026), and albuterol (n = 976). Two deaths were reported, 1 each in the formoterol (asthma exacerbation) and the placebo (hemorrhagic pancreatitis) groups. No statistically significant differences in serious asthma exacerbations were observed compared with placebo in adolescents and adults. In children, a higher frequency of hospitalizations was observed among patients treated with formoterol compared with placebo (OR 8.4; 95% CI: 1.1-65.3). A trend toward fewer exacerbations was observed among subjects reporting concomitant ICS use at baseline Paracetamol Pediatric Dose .

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To present the case of a patient with persistent bronchospasm, refractory to treatment with β2-agonists, that resolved promptly with continuous inhalation of large dose (1000 mcg/hr) ipratropium bromide, and to discuss the possibility of tolerance to β2-agonists as the cause for his failure to respond to adrenergic medications. The patient had received multiple doses of albuterol, as well as subcutaneous terbutaline (0.3 mg), intravenous magnesium sulfate (1 g) and intravenous dexamethasone (10 mg) prior to his admission to the intensive care unit. He remained symptomatic despite systemic intravenous steroids, continuous intravenous terbutaline (up to 0.6 mcg/kg/min), and continuous nebulized albuterol (up to 20 mg/hr for 57 hr) followed by 49 hours of continuous levalbuterol (7 mg/hr). Due Drug Zantac to the lack of response, all β2-agonists were discontinued at 106 hours post-admission, and he was started on large dose ipratropium bromide (1000 mcg/hr) by continuous nebulization. Clinical improvement was evident within 1 hour and complete resolution of his symptoms within 4 hours. Continuous inhalation of large dose ipratropium bromide may be an effective regimen for the treatment of patients hospitalized with acute asthma who are deemed to be nonresponsive and/or tolerant to β2-agonist therapy.

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The goal of this study was to examine the impact of a multidisciplinary intervention designed to improve appropriate albuterol inhaler utilization among patients with asthma.

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4-Hydroxy-2-nonenal (4-HNE) is a major reactive aldehyde formed by lipid peroxidation, and it plays an important role in the pathogenesis of several vascular diseases, including diabetes mellitus. In this study, we examined the effects of 4-HNE on the vasodilatory mechanisms of rat retinal arterioles. The retinal vasodilator responses were assessed by measuring the diameter of retinal arterioles in the fundus images. Intravitreal injection of 4-HNE significantly prevented the vasodilation of retinal arterioles induced by the β2-adrenoceptor agonist salbutamol but not the nitric oxide donor (±)-(E)-4-ethyl-2-[(E)-hydroxyimino]-5-nitro-3-hexenamide (NOR3). Iberiotoxin, an inhibitor of large-conductance KCa (BKCa) channels, significantly reduced the salbutamol-induced vasodilation of retinal arterioles. The vasodilator effect of BMS-191011, a BKCa channel opener, on retinal arterioles was significantly attenuated by 4-HNE. These results suggest that 4-HNE attenuates retinal vasodilator responses to β2-adrenoceptor agonists through the impairment of the BKCa pathway. The direct effect of 4-HNE on retinal blood vessels may, therefore, contribute to the retinal vascular dysfunction observed in patients with diabetes mellitus.